Mannose-Binding Lectin Deficiency Is Associated with Myocardial Infarction: The HUNT2 Study in Norway

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Standard

Mannose-Binding Lectin Deficiency Is Associated with Myocardial Infarction : The HUNT2 Study in Norway. / Vengen, Inga Thorsen; Madsen, Hans O; Garred, Peter; Platou, Carl; Vatten, Lars; Videm, Vibeke.

In: P L o S One, Vol. 7, No. 7, 2012, p. e42113.

Research output: Contribution to journalJournal articleResearchpeer-review

Harvard

Vengen, IT, Madsen, HO, Garred, P, Platou, C, Vatten, L & Videm, V 2012, 'Mannose-Binding Lectin Deficiency Is Associated with Myocardial Infarction: The HUNT2 Study in Norway', P L o S One, vol. 7, no. 7, pp. e42113. https://doi.org/10.1371/journal.pone.0042113

APA

Vengen, I. T., Madsen, H. O., Garred, P., Platou, C., Vatten, L., & Videm, V. (2012). Mannose-Binding Lectin Deficiency Is Associated with Myocardial Infarction: The HUNT2 Study in Norway. P L o S One, 7(7), e42113. https://doi.org/10.1371/journal.pone.0042113

Vancouver

Vengen IT, Madsen HO, Garred P, Platou C, Vatten L, Videm V. Mannose-Binding Lectin Deficiency Is Associated with Myocardial Infarction: The HUNT2 Study in Norway. P L o S One. 2012;7(7):e42113. https://doi.org/10.1371/journal.pone.0042113

Author

Vengen, Inga Thorsen ; Madsen, Hans O ; Garred, Peter ; Platou, Carl ; Vatten, Lars ; Videm, Vibeke. / Mannose-Binding Lectin Deficiency Is Associated with Myocardial Infarction : The HUNT2 Study in Norway. In: P L o S One. 2012 ; Vol. 7, No. 7. pp. e42113.

Bibtex

@article{1a695bc7f33c4957bdd94676a5ca5106,
title = "Mannose-Binding Lectin Deficiency Is Associated with Myocardial Infarction: The HUNT2 Study in Norway",
abstract = "Mannose-binding lectin (MBL) and ficolins activate the complement cascade, which is involved in atherogenesis. Based on a pilot study, we hypothesized that functional polymorphisms in the MBL gene (MBL2) leading to dysfunctional protein are related to development of myocardial infarction (MI). The aim of the present study was to study polymorphisms in MBL2 and ficolin genes in relation to the risk of MI.",
author = "Vengen, {Inga Thorsen} and Madsen, {Hans O} and Peter Garred and Carl Platou and Lars Vatten and Vibeke Videm",
year = "2012",
doi = "10.1371/journal.pone.0042113",
language = "English",
volume = "7",
pages = "e42113",
journal = "PLoS ONE",
issn = "1932-6203",
publisher = "Public Library of Science",
number = "7",

}

RIS

TY - JOUR

T1 - Mannose-Binding Lectin Deficiency Is Associated with Myocardial Infarction

T2 - The HUNT2 Study in Norway

AU - Vengen, Inga Thorsen

AU - Madsen, Hans O

AU - Garred, Peter

AU - Platou, Carl

AU - Vatten, Lars

AU - Videm, Vibeke

PY - 2012

Y1 - 2012

N2 - Mannose-binding lectin (MBL) and ficolins activate the complement cascade, which is involved in atherogenesis. Based on a pilot study, we hypothesized that functional polymorphisms in the MBL gene (MBL2) leading to dysfunctional protein are related to development of myocardial infarction (MI). The aim of the present study was to study polymorphisms in MBL2 and ficolin genes in relation to the risk of MI.

AB - Mannose-binding lectin (MBL) and ficolins activate the complement cascade, which is involved in atherogenesis. Based on a pilot study, we hypothesized that functional polymorphisms in the MBL gene (MBL2) leading to dysfunctional protein are related to development of myocardial infarction (MI). The aim of the present study was to study polymorphisms in MBL2 and ficolin genes in relation to the risk of MI.

U2 - 10.1371/journal.pone.0042113

DO - 10.1371/journal.pone.0042113

M3 - Journal article

C2 - 22848725

VL - 7

SP - e42113

JO - PLoS ONE

JF - PLoS ONE

SN - 1932-6203

IS - 7

ER -

ID: 48448976