Goodpasture's syndrom. Antiglomerulaer basalmembranantisofmedieret glomerulonephritis

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Standard

Goodpasture's syndrom. Antiglomerulaer basalmembranantisofmedieret glomerulonephritis. / Pagsberg, K; Pedersen, G; Hansen, F M.

I: Ugeskrift for Laeger, Bind 151, Nr. 34, 21.08.1989, s. 2141-4.

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning

Harvard

Pagsberg, K, Pedersen, G & Hansen, FM 1989, 'Goodpasture's syndrom. Antiglomerulaer basalmembranantisofmedieret glomerulonephritis', Ugeskrift for Laeger, bind 151, nr. 34, s. 2141-4.

APA

Pagsberg, K., Pedersen, G., & Hansen, F. M. (1989). Goodpasture's syndrom. Antiglomerulaer basalmembranantisofmedieret glomerulonephritis. Ugeskrift for Laeger, 151(34), 2141-4.

Vancouver

Pagsberg K, Pedersen G, Hansen FM. Goodpasture's syndrom. Antiglomerulaer basalmembranantisofmedieret glomerulonephritis. Ugeskrift for Laeger. 1989 aug. 21;151(34):2141-4.

Author

Pagsberg, K ; Pedersen, G ; Hansen, F M. / Goodpasture's syndrom. Antiglomerulaer basalmembranantisofmedieret glomerulonephritis. I: Ugeskrift for Laeger. 1989 ; Bind 151, Nr. 34. s. 2141-4.

Bibtex

@article{3d64905dd9664b008580fed28ce4b669,
title = "Goodpasture's syndrom. Antiglomerulaer basalmembranantisofmedieret glomerulonephritis",
abstract = "A review is presented of antiglomerular basal membrane antibody-mediated glomerulonephritis (anti-GBM-Ab-nephritis) which constitutes 2-5% of all cases of acute glomerulonephritis. The disease frequently commences in the age group 20-30 years but may be encountered in all age groups, in women particularly at 60 years of age. The disease is due to autoantibodies (IgG) to the basal membranes in the glomeruli and alveoli. Deposition of IgG with C3 precipitates an inflammatory reaction which causes renal and possibly also pulmonary damage. It is possible to demonstrate anti-GMB-antibodies in the blood and, by means of immunofluorescence microscopy, these and C3 may be demonstrated in the basal membranes in the glomeruli and alveoli. The disease is still serious but introduction of immune-suppressive treatment and plasmapheresis has improved the prognosis considerably.",
author = "K Pagsberg and G Pedersen and Hansen, {F M}",
year = "1989",
month = aug,
day = "21",
language = "Dansk",
volume = "151",
pages = "2141--4",
journal = "Ugeskrift for Laeger",
issn = "0041-5782",
publisher = "Almindelige Danske Laegeforening",
number = "34",

}

RIS

TY - JOUR

T1 - Goodpasture's syndrom. Antiglomerulaer basalmembranantisofmedieret glomerulonephritis

AU - Pagsberg, K

AU - Pedersen, G

AU - Hansen, F M

PY - 1989/8/21

Y1 - 1989/8/21

N2 - A review is presented of antiglomerular basal membrane antibody-mediated glomerulonephritis (anti-GBM-Ab-nephritis) which constitutes 2-5% of all cases of acute glomerulonephritis. The disease frequently commences in the age group 20-30 years but may be encountered in all age groups, in women particularly at 60 years of age. The disease is due to autoantibodies (IgG) to the basal membranes in the glomeruli and alveoli. Deposition of IgG with C3 precipitates an inflammatory reaction which causes renal and possibly also pulmonary damage. It is possible to demonstrate anti-GMB-antibodies in the blood and, by means of immunofluorescence microscopy, these and C3 may be demonstrated in the basal membranes in the glomeruli and alveoli. The disease is still serious but introduction of immune-suppressive treatment and plasmapheresis has improved the prognosis considerably.

AB - A review is presented of antiglomerular basal membrane antibody-mediated glomerulonephritis (anti-GBM-Ab-nephritis) which constitutes 2-5% of all cases of acute glomerulonephritis. The disease frequently commences in the age group 20-30 years but may be encountered in all age groups, in women particularly at 60 years of age. The disease is due to autoantibodies (IgG) to the basal membranes in the glomeruli and alveoli. Deposition of IgG with C3 precipitates an inflammatory reaction which causes renal and possibly also pulmonary damage. It is possible to demonstrate anti-GMB-antibodies in the blood and, by means of immunofluorescence microscopy, these and C3 may be demonstrated in the basal membranes in the glomeruli and alveoli. The disease is still serious but introduction of immune-suppressive treatment and plasmapheresis has improved the prognosis considerably.

M3 - Tidsskriftartikel

VL - 151

SP - 2141

EP - 2144

JO - Ugeskrift for Laeger

JF - Ugeskrift for Laeger

SN - 0041-5782

IS - 34

ER -

ID: 34084849