Paracetamol-induced acute kidney injury in the absence of acute liver injury: a retrospective cohort study

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Standard

Paracetamol-induced acute kidney injury in the absence of acute liver injury : a retrospective cohort study. / Daoud, Alaa; Dalhoff, Kim Peder; Petersen, Tonny Studsgaard.

I: Toxicology Communications, Bind 6, Nr. 1, 2022, s. 97-100.

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt

Harvard

Daoud, A, Dalhoff, KP & Petersen, TS 2022, 'Paracetamol-induced acute kidney injury in the absence of acute liver injury: a retrospective cohort study', Toxicology Communications, bind 6, nr. 1, s. 97-100. https://doi.org/10.1080/24734306.2022.2117941

APA

Daoud, A., Dalhoff, K. P., & Petersen, T. S. (2022). Paracetamol-induced acute kidney injury in the absence of acute liver injury: a retrospective cohort study. Toxicology Communications, 6(1), 97-100. https://doi.org/10.1080/24734306.2022.2117941

Vancouver

Daoud A, Dalhoff KP, Petersen TS. Paracetamol-induced acute kidney injury in the absence of acute liver injury: a retrospective cohort study. Toxicology Communications. 2022;6(1):97-100. https://doi.org/10.1080/24734306.2022.2117941

Author

Daoud, Alaa ; Dalhoff, Kim Peder ; Petersen, Tonny Studsgaard. / Paracetamol-induced acute kidney injury in the absence of acute liver injury : a retrospective cohort study. I: Toxicology Communications. 2022 ; Bind 6, Nr. 1. s. 97-100.

Bibtex

@article{3709c1baf0874842a8f006654db6f101,
title = "Paracetamol-induced acute kidney injury in the absence of acute liver injury: a retrospective cohort study",
abstract = "Paracetamol-induced nephrotoxicity in the absence of hepatotoxicity is only described in a few studies. The aim of this retrospective cohort study with data from the Capital Region of Denmark 2010-2017, was to examine the incidence of possible paracetamol-induced nephrotoxicity in absence of hepatotoxicity. Only one out of 5,827 admissions (0.02%) included in the study developed clinically relevant acute kidney injury (AKI) that could be attributed to paracetamol in absence of acute liver injury. This study demonstrates that clinically relevant AKI due to paracetamol overdose rarely occurs without concomitant hepatic injury when excluding other prerenal, renal, and postrenal causes of renal dysfunction, NAC interference and chronic kidney injury.",
author = "Alaa Daoud and Dalhoff, {Kim Peder} and Petersen, {Tonny Studsgaard}",
year = "2022",
doi = "10.1080/24734306.2022.2117941",
language = "English",
volume = "6",
pages = "97--100",
journal = "Toxicology Communications",
issn = "2473-4306",
publisher = "Taylor & Francis",
number = "1",

}

RIS

TY - JOUR

T1 - Paracetamol-induced acute kidney injury in the absence of acute liver injury

T2 - a retrospective cohort study

AU - Daoud, Alaa

AU - Dalhoff, Kim Peder

AU - Petersen, Tonny Studsgaard

PY - 2022

Y1 - 2022

N2 - Paracetamol-induced nephrotoxicity in the absence of hepatotoxicity is only described in a few studies. The aim of this retrospective cohort study with data from the Capital Region of Denmark 2010-2017, was to examine the incidence of possible paracetamol-induced nephrotoxicity in absence of hepatotoxicity. Only one out of 5,827 admissions (0.02%) included in the study developed clinically relevant acute kidney injury (AKI) that could be attributed to paracetamol in absence of acute liver injury. This study demonstrates that clinically relevant AKI due to paracetamol overdose rarely occurs without concomitant hepatic injury when excluding other prerenal, renal, and postrenal causes of renal dysfunction, NAC interference and chronic kidney injury.

AB - Paracetamol-induced nephrotoxicity in the absence of hepatotoxicity is only described in a few studies. The aim of this retrospective cohort study with data from the Capital Region of Denmark 2010-2017, was to examine the incidence of possible paracetamol-induced nephrotoxicity in absence of hepatotoxicity. Only one out of 5,827 admissions (0.02%) included in the study developed clinically relevant acute kidney injury (AKI) that could be attributed to paracetamol in absence of acute liver injury. This study demonstrates that clinically relevant AKI due to paracetamol overdose rarely occurs without concomitant hepatic injury when excluding other prerenal, renal, and postrenal causes of renal dysfunction, NAC interference and chronic kidney injury.

U2 - 10.1080/24734306.2022.2117941

DO - 10.1080/24734306.2022.2117941

M3 - Journal article

VL - 6

SP - 97

EP - 100

JO - Toxicology Communications

JF - Toxicology Communications

SN - 2473-4306

IS - 1

ER -

ID: 345641609